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2025 ICD-10-CM code A84

Tick-borne viral encephalitis. Includes: tick-borne viral meningoencephalitis.

Use additional codes to identify any neurological complications or sequelae resulting from TBE. If known, specify the subtype of TBE (European, Siberian, or Far Eastern) using an appropriate external cause code.

Medical necessity for TBE treatment is established by the presence of signs and symptoms consistent with the infection, as well as laboratory confirmation of the virus or specific antibodies. The medical record should document the clinical presentation, diagnostic findings, and the need for supportive care, hospitalization, or intensive care based on the severity of the illness.

Clinicians diagnose TBE based on patient history (including travel to endemic areas and possible tick exposure), physical and neurological examinations, and laboratory tests. Blood tests and lumbar puncture (spinal tap) are used to identify the virus or antibodies in bodily fluids. Brain imaging studies like CT or MRI can aid in differential diagnosis and assess the extent of neurological involvement. Treatment is primarily supportive, focusing on managing symptoms and providing intensive care for severe cases.

In simple words: Tick-borne encephalitis (TBE) is a disease spread by ticks that causes swelling in the brain. It's found in some parts of Europe and Asia. You can get it from a tick bite or by drinking raw milk from infected animals. Most people have no symptoms or mild flu-like symptoms. Some people get more serious problems like headaches, stiff neck, confusion, weakness, and even seizures. There's no specific medicine for TBE, so doctors focus on treating the symptoms.

Tick-borne encephalitis (TBE) is a viral infection affecting the central nervous system. It is transmitted primarily through the bite of infected ticks, predominantly found in woodland habitats across parts of Europe and Asia. TBE can also be contracted through the consumption of unpasteurized dairy products, particularly from infected goats, sheep or cows. The virus can manifest in two phases. The initial phase presents with non-specific symptoms such as fever, fatigue, headache, muscle aches (myalgia), and nausea. The second phase, occurring in a subset of patients, involves neurological manifestations like meningitis (inflammation of the membranes surrounding the brain and spinal cord) and/or encephalitis (inflammation of the brain). These can lead to a range of neurological complications, including memory problems, cognitive impairment, muscle weakness, tremors, impaired coordination, involuntary movements, and sensory disturbances like numbness or tingling.

Example 1: A hiker returning from a trip to Central Europe presents with fever, headache, and fatigue. Blood tests and a lumbar puncture confirm TBE., A child develops a stiff neck, confusion, and seizures after consuming unpasteurized goat milk while visiting family in an Eastern European country. Laboratory findings indicate TBE., A forestry worker in Siberia experiences fever, muscle aches, and nausea. After a period of apparent recovery, they develop neurological symptoms including meningitis and encephalitis. TBE is confirmed through CSF analysis.

Documentation for TBE should include details of symptom onset, travel history, potential tick exposure, physical and neurological examination findings, laboratory results (blood tests, lumbar puncture), and imaging studies (CT, MRI). Details of supportive treatment and management of complications should also be recorded.

** No specific antiviral therapy is available for TBE.Treatment is supportive, focusing on managing symptoms, including pain and fever. Prevention methods include vaccination, protective clothing and repellents and avoiding the consumption of unpasteurized dairy products.

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iFrame™ AI's knowledge is aligned with and limited to the materials uploaded by users and should not be interpreted as medical, legal, or any other form of advice by iFrame™.